A oftalmopatia tireoidea é uma doença autoimune da órbita intimamente associada com o hipertireoidismo (doença de Graves), embora ambas as condições. Do ponto de vista clínico, a doença de Graves caracteriza-se por aumento difuso e hiperatividade da glândula tireóide, associada ou não a oftalmopatia. Oftalmopatia tireoidea revisitada*. Thyroid A oftalmopatia tireoidea é doença orbitária autoimune intimamente com o hipertireoidismo (doença de Graves).

Author: Shaktilabar Moogut
Country: Tanzania
Language: English (Spanish)
Genre: Science
Published (Last): 27 August 2004
Pages: 148
PDF File Size: 16.72 Mb
ePub File Size: 11.23 Mb
ISBN: 910-1-82930-420-5
Downloads: 72451
Price: Free* [*Free Regsitration Required]
Uploader: Faurg

Graves’ disease in children and adolescents: Endocrinol Metab Clin North Am.

Oftalkopatia HB, Wartofsky L. Current and emerging treatment options for Graves’ hyperthyroidism. Prediction of cure and risk of hypothyroidism in patients receiving I for hyperthyroidism.

Sabino de Arana, 40, 3. Agranulocytosis induced by propylthiouracil: The efficacy of thyroidectomy for Graves’ disease: Interferon-alfa induced thyroid dysfunction: Hypothyroidism as a late sequela in patient with Graves’ disease treated with antithyroid agents. Treatment for childhood-onset Graves’ disease in Japan: Clin Endocrinol Oxf50pp. Incidence and risk factors for agranulocytosis in Latin American countries–the Ooftalmopatia Study: Occurrence of ophthalmopathy after treatment for Graves’ hyperthyroidism.


Clin Endocrinol Oxf42pp.

Thyroid ophthalmopathy revisited

Five-year follow-up of percutaneous oftal,opatia injection for the treatment of hyperfunctioning thyroid nodules: Controversies in the clinical evaluation of active thyroid-associated orbitopathy: Propranolol as an adjunct therapy for hyperthyroid tremor.

Graves’ ophthalmopathy is considered to be an autoimmune disorder and a component of Graves’ disease, and represents a process confined to the orbit where the connective tissues become inflamed and accumulate glycosaminoglycans.

Graves’ disease is the most frequent cause of hyperthyroidism. Using morphologic parameters of extraocular muscles for diagnosis and follow-up of Graves’ ophthalmo pathy: Clin Endocrinol Oxf45pp.

Resumption of methimazole after I therapy of hyperthyroid diseases: Testicular function after I therapy for hyperthyroidism. Surgical management of Graves’ diseaseyear prospective trial at a single institution. Treatment of hyperthyroidism in infancy and adolescence.

Clinical research into Graves’ ophthalmopathy has been hampered by its characteristic multiphasic natural history. Nat Clin Pract Endocrinol Metab.

Use of corticosteroids to prevent progression of Graves’ ophthalmopathy after radioiodine therapy for hyperthyroidism. Rajatanavi R, Braverman LE.


Dose-response study on thyrotoxic patients undergoing positron emission tomography and radioiodine therapy. Intravenous methimazole in the treatment of refractory hyperthyroidism.

Risk of malignancies in non-palpable thyroid nodules: Cardiomyopathy associated with Graves’ disease. Endocrinol Metab Clin North Am ; MR imaging of the orbits. Evaluation of diagnosis and treatment results in children with Graves’ disease with emphasis on the pubertal status of patients.

Radiation and Graves’ ophthalmopathy. Surgery is now rarely performed. The effect of diltiazem on the manifestations of hyperthyroidism oftalmipatia thyroid function tests.

High pre-therapy [99mTc]pertechnetate thyroid uptake, thyroid size and thyrostatic drugs: N Engl J Med. Color flow Doppler sonography rapidly ofatlmopatia type I and type II amiodarone-induced thyrotoxicosis.

The Multicenter Study Group. Thionamida therapy in Graves’ disease: Rivkees SA, Dinauer C. Hell J Nucl Med.